Study of Atherosclerotic disease & spontaneous Venous Thrombosis

All patients in the poll were evaluated by ultrasonography as suffering from symptomatic proximal-vein thrombosis. These patients were selected from those admitted to install where Prandoni and colleagues work. Control subjects were selected randomly from patients admitted for reasons not related to venous thromboembolism or atherosclerosis. Statistics were analyzed using the Kruskal-Wallis test for continuous variables and the chi-square test for dichotomous variables.

The researchers shut that figure outivation of the coagulation system may act as a pathophysiological link between atherosclerosis and venous thrombosis. The presence of carotid plaques was significantly higher in those patients with thrombotic events that were unexplained. The researchers avoided predetermine in the film by evaluating the presence of risk factors and fashioning records on standard forms before ultrasonography was undertaken.

Validity of the study was reinforced by establishing the reproducibility of ultrasonographic assessment of carotid plaques in a subgroup of 100 patients (Prandoni, et al., 2003). Validity was also established by making adjustments to the confounding effects of risk factors. The study cannot conclude a causative role or atherosclerosis with applaud to venous thromboembolism, but it does highlight a potential family relationship between arterial and venous disorders.

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